CHEMOKINE RECEPTOR GENE CRUCIAL IN THE CONTROL OF TUBERCULOSIS, GLADSTONE/UCSF
Controlling a tuberculosis infection requires precise interactions between a number of different immune cells. Researchers are showing for the first time that the absence of the gene for CCR2, a receptor on white blood cells known to be important for cell migration, can have fatal consequences in this setting.
The study is a joint collaboration between the Gladstone Institute of Cardiovascular Disease and the UCSF Division of Infectious Diseases, published in PNAS
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